INFECTION IN SYSTEMIC LUPUS ERYTHEMATOSUS
Identifieur interne : 002756 ( Main/Exploration ); précédent : 002755; suivant : 002757INFECTION IN SYSTEMIC LUPUS ERYTHEMATOSUS
Auteurs : Michelle PetriSource :
- Rheumatic Disease Clinics of North America [ 0889-857X ] ; 1998.
English descriptors
- Teeft :
- Andonopoulos, Arch intern, Arthritis, Arthritis rheum, Autoantibody, Bacteremia, Candida, Carinii, Cerebrospinal, Chemotactic, Clin, Coccidioidomycosis, Cohort, Coli, Corticosteroid, Cyclophosphamide, Disease activity, Dysfunction, Erythematosus, Ginzler, Herpes, Herpes zoster, Immune, Immunodeficiency, Immunol, Immunosuppressive, Infection, Infection rate, Klebsiella, Leukocyte, Leukopenia, Lupus, Lupus erythematosus, Lupus nephritis, Lymphocyte, Major infections, Massardo, Nephritis, Neutrophil, Nived, Opportunistic, Opportunistic infections, Pathogen, Petri, Phagocytosis, Pneumococcal, Pneumococcus, Pneumocystis, Polymorphonuclear leukocytes, Predictor, Prospective study, Receptor, Renal, Renal failure, Rheum, Rheumatoid, Rheumatoid arthritis, Rheumatol, Risk factors, Salmonella, Semin arthritis rheum, Septic arthritis, Staple, Systemic, Systemic lupus erythematosus, Toxoplasmosis, Wong, Yuhara, Zoster.
Abstract
The improved survival of systemic lupus erythematosus (SLE) patients since the 1950s is the result of not only better treatment of SLE, but also supportive treatment of renal failure (transplant and dialysis) and the wealth of antibiotics now available. Ironically, the wider use of immunosuppressives, especially the alkylating drugs, and the longer survival of patients with renal insufficiency and renal failure have made the identification and appropriate treatment of infection in SLE an on-going challenge. It has always seemed extremely ironic that SLE patients have an increased risk for infection, given that they exhibit so many signs of B-cell hyperactivity. SLE patients, after all, are characterized by having hypergammaglobulinemia and elevated antibody titers to multiple viruses and other pathogens. It is this paradoxthat SLE patients, whose immune systems attack themselves, are often unable to mount an effective attack against outside agentsthat is explored in this article.
Url:
DOI: 10.1016/S0889-857X(05)70016-8
Affiliations:
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Ironically, the wider use of immunosuppressives, especially the alkylating drugs, and the longer survival of patients with renal insufficiency and renal failure have made the identification and appropriate treatment of infection in SLE an on-going challenge. It has always seemed extremely ironic that SLE patients have an increased risk for infection, given that they exhibit so many signs of B-cell hyperactivity. SLE patients, after all, are characterized by having hypergammaglobulinemia and elevated antibody titers to multiple viruses and other pathogens. It is this paradoxthat SLE patients, whose immune systems attack themselves, are often unable to mount an effective attack against outside agentsthat is explored in this article.</div></front></TEI><affiliations><list></list><tree><noCountry><name sortKey="Petri, Michelle" sort="Petri, Michelle" uniqKey="Petri M" first="Michelle" last="Petri">Michelle Petri</name></noCountry></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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